| Publication information |
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Source: A Text Book on Uric Acid and Its Congeners Source type: book Document type: book chapter Document title: “Traumatisms and Surgical Notes” [chapter 21] Author(s): Gilbert, George Abner Publisher: Danbury Medical Printing Company Place of publication: Danbury, Connecticut Year of publication: 1907 Pagination: 188-96 (excerpt below includes only pages 194-95) |
| Citation |
| Gilbert, George Abner. “Traumatisms and Surgical Notes” [chapter 21]. A Text Book on Uric Acid and Its Congeners. Danbury: Danbury Medical Printing, 1907: pp. 188-96. |
| Transcription |
| excerpt of chapter |
| Keywords |
| William McKinley (recovery: speculation); William McKinley (medical condition). |
| Named persons |
| William McKinley. |
| Notes |
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From title page: A Text Book on Uric Acid and Its Congeners, with
Special Reference to Its Physical and Chemical Properties, Its Metabolism
and Accumulation in the Organism, Together with the Disease Processes
Arising Therefrom and Their Ætiological Therapy; for Medical Students
and Practitioners.
From title page: By George Abner Gilbert, M. D., Member of Local, County and State Medical Societies of Connecticut; Physician to the Danbury Hospital, etc.; Once Fellow of the New York Academy of Medicine. |
| Document |
Traumatisms and Surgical Notes [excerpt]
It was the generally accepted belief
among the members of the profession, that had President McKinley (at the time
of the wound and operation which proved so disastrous) been in the prime and
vigor of health, with organs of secretion and excretion equal to the full performance
of their duty, the wound and operation might not have proven fatal. It is believed
that the impairment of the metabolic functions and gradual accumulation of toxic
waste products within the system served to paralyze reparative energy when the
occasion demanded. The necessity of immediate operation rendered it impossible
in this instance to rid the organism of purin waste in the manner recommended
in such cases.
The local irritation of tissue observed in the
diabetic patient, when injured or operated upon, is well known to the surgeon.
The frequent association of gout and diabetes has caused much speculation as
to the probable similarity of origin of these two complaints—though one represents
faulty metabolism of nitrogenous products and the other of the carbo-hydrates;
and much food for reflection is afforded the thoughtful physician in such cases
as [194][195] that of the lamented McKinley. Was
he suffering from purin excess (or the so-called “gouty diathesis”), and were
retrograde tissue products present in such quantity as to serve as a disturbing
factor?