Publication information |
Source: Merck’s Archives of Materia Medica and Drug Therapy Source type: journal Document type: letter to the editor Document title: “The Late President McKinley” Author(s): Williams, George A. Date of publication: October 1901 Volume number: 3 Issue number: 10 Pagination: 411 |
Citation |
Williams, George A. “The Late President McKinley.” Merck’s Archives of Materia Medica and Drug Therapy Oct. 1901 v3n10: p. 411. |
Transcription |
full text |
Keywords |
William McKinley (medical condition); William McKinley (death, cause of). |
Named persons |
George A. Williams. |
Document |
The Late President McKinley
M
In the President’s case the surgeons were remarkably
expert and the operation was most complete and thoroughly scientific; but no
doubt the injury was fatal from the beginning. As one prominent pathologist
stated, “the President’s nervous system was constantly toned up to the highest
pitch, and in receiving this severe injury there was a lack of nerve force to
carry the patient through the crisis.” The constantly rapid and feeble pulse
was a sure indication of severe nervous prostration, and judging from what we
have seen, the patient never fully recovered from the “shock” attending the
injury. It is well known that gangrene is not idiopathic, but an effect which
may be produced by various causes, and is not necessarily produced by gunshot
wounds. During the war, as we have seen, gangrene was quite common where the
men were exhausted from long marches, poor food, and exposure, and it is well
known, that a gunshot wound or any kind of wound, is liable to produce gangrene
in any case of severe prostration or extreme debility. How well we see this
principle illustrated in old, debilitated subjects: if an operation be performed
in such a case, as a rule, the wound shows no disposition to heal, gangrene
ensues, and the patient dies, not from gangrene, but from “shock” and nerve
exhaustion. . . . In the President’s case we had, first,
the severe shock, then the feeble pulse with other impaired physiological action,
and then gangrene and collapse, following complete nerve exhaustion. May we
not, then, regard “shock” as the true cause of death in this case? Inasmuch
as we are all anxious for truth and enlightenment, the writer will be pardoned
for not allowing this case to pass unnoticed, without some comment.
G
Bay City, Mich.