Publication information |
Source: International Journal of Surgery Source type: journal Document type: editorial Document title: “Results of the Autopsy in President McKinley’s Case” Author(s): anonymous Date of publication: November 1901 Volume number: 14 Issue number: 11 Pagination: 343 |
Citation |
“Results of the Autopsy in President McKinley’s Case.” International Journal of Surgery Nov. 1901 v14n11: p. 343. |
Transcription |
full text |
Keywords |
William McKinley (medical condition); William McKinley (death, cause of); William McKinley (medical care: personal response). |
Named persons |
Hans Chiari; Simon Flexner; Herbert M. Hill; Herman G. Matzinger. |
Document |
Results of the Autopsy in President McKinley’s Case
From the standpoint of pathological anatomy,
the report of the autopsy performed on the body of our late president fails
to entirely account for the production of the necrotic changes observed in the
track of the missile. In the report of the bacteriological examination made
by Dr. Matzinger, the bacteriologist to the New York State Pathological Laboratory,
of whose high competence in such matters there is no question, it is shown that
there were no known pathogenic bacteria in the necrotic cavity, warranting the
statement of Dr. Matzinger to the effect that “bacterial infection was not a
factor in the production of the conditions found at the autopsy.” The bacteriological
and chemical examinations of the chambers and barrel of the revolver, as well
as of the empty shells and cartridges, ordered by the district attorney, was
entirely negative, excepting that from a loaded cartridge there was grown an
ordinary staphylococcus and a mold. The chemical examination made by Dr. Hill,
the chemist, was also negative. This goes very far to prove that the theory
of a poisoned bullet may now be entirely discarded. We are, therefore, in a
position to state that from the bacteriological and chemical standpoint there
were no factors to be found that might have served to explain the presence of
the peculiar necrotic area observed at the autopsy, and that we must seek further
for an explanation of the occurrence of this unusual phenomenon. Such an explanation
must evidently be at best but an unsatisfactory one from the point of view of
a rigorous scientific accuracy, since we are thrown back more or less upon theoretical
notions derived in part from the appearance of the lesions directly caused by
the bullet, and in part from the observation of the other organs of the body.
In the first place the report makes it evident that there was no general infection
of a pyemic or septicemic nature. The blood taken from the heart was sterile,
and cultures from the peritoneal surfaces showed that no peritonitis existed.
Neither was there, in any part of the track of the bullet, any collection of
pus. The heart was rather small, its walls thin and flaccid, and in a condition
of fatty degeneration, infiltration and brown atrophy. The kidneys were in a
condition of partial parenchymatous degeneration. These organs were, therefore,
affected in a manner such as to very distinctly diminish the force of resistance
to injury possessed by the patient, and to sufficiently explain the lack of
reparative action manifested by the findings of the autopsy. Experiments by
Flexner and Chiari, mentioned in the report, have shown that animals seem to
have died as a result of not very extensive lesions inflicted upon the pancreas,
and that concussions and slight injuries of this organ may be a factor in the
development of necrosis. The latter writer observed, although it is a comparatively
rare condition, extensive areas of softening and necrosis of the pancreas, especially
of the posterior central portion which lies directly over the bodies of the
vertebræ, where the organ is most exposed to pressure or the effects of concussion.
Since the injury inflicted upon the left kidney was of a comparatively slight
extent, we must believe that the lesion of the pancreas, assisted by the impaired
condition of the heart and kidneys, was the chief and determining cause of the
lamentable end of the distinguished patient. While it is unfortunate that our
knowledge of the lesions of the pancreas should be so slight, this case seems
to prove that injuries of this organ, especially in patients already somewhat
debilitated by other pathological conditions, are an additional source of danger
in the chapter of abdominal traumatisms.
The reading of the exhaustive report upon the
late president’s case leaves the reader under the firm impression that nothing
that might have been achieved by the most painstaking care and the application
of the most thorough scientific knowledge was neglected in any manner whatsoever.